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In nerves uncovered to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose additional power substrate is required when the tissue energy demand increases. Such information are essential as they show that glycogen has an essential position underneath physiological conditions. The truth that glycogen supported CAP conduction within the absence of exogenously utilized glucose in all probability has no worth in ascribing a task to glycogen, since aglycaemia is an artificial experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus can't be glycogen’s major function. However supplying lactate to axons when tissue energy demand will increase implies a novel position for brain glycogen and suggests it acts as an power buffer rather than an vitality reserve, whose function is not to single-handedly support conduction, but to act in live performance with ambient ranges of normoglycemic glucose.
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