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In nerves exposed to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose additional power substrate is required when the tissue vitality demand increases. Such data are necessary as they show that glycogen has an vital role under physiological conditions. The truth that glycogen supported CAP conduction in the absence of exogenously applied glucose most likely has no value in ascribing a job to glycogen, since aglycaemia is an synthetic experimental process, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus can't be glycogen’s most important role. However supplying lactate to axons when tissue energy demand increases implies a novel role for brain glycogen and suggests it acts as an power buffer somewhat than an energy reserve, whose position is not to single-handedly assist conduction, however to act in concert with ambient ranges of normoglycemic glucose.
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